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"If you have knowledge, let others light their candles in it." |
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For Dentists: Topics Related to Endodontics |
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Diagnosing Pain
○ Infected Root Canal Strategies - 2001 CDA |
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| ▒ Patient Registration |
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I. Management at Site of Injury Replant immediately, if possible. If contaminated, rinse with water before replanting. When immediate replantation is not possible, place tooth in the best transport medium available If none of the above is readily available, use water. III. Management in the Dental Office B. Management of the Root Surface
Tightly suture any soft tissue lacerations, particularly in the cervical region. E. Splinting (indicated in most cases) IV. Adjunctive Drug Therapy Considerations A. Tooth with open apex (divergent apex) and less than 2 hours extraoral dry time: B. Tooth with open apex (divergent apex) and greater than 2 hours extraoral dry time: C. Tooth with partially to completely closed apex and less than 2 hours extraoral dry time: D. Tooth with partially to completely closed apex and greater than 2 hours extraoral dry time:
VI. Restoration of the Avulsed Tooth A. Recommended Temporary Restorations (placed prior to final obturation) B. Recommended Permanent Restorations (placed immediately after final obturation) Vll. Additional Considerations
Tightly suture any soft tissue lacerations, particularly in the cervical region. E. Splinting (indicated in most cases) VIII. Adjunctive Drug Therapy Considerations A. Tooth with open apex (divergent apex) and less than 2 hours extraoral dry time: B. Tooth with open apex (divergent apex) and greater than 2 hours extraoral dry time: C. Tooth with partially to completely closed apex and less than 2 hours extraoral dry time: D. Tooth with partially to completely closed apex and greater than 2 hours extraoral dry time:
V. Restoration of the Avulsed Tooth A. Recommended Temporary Restorations (placed prior to final obturation) B. Recommended Permanent Restorations (placed immediately after final obturation) (Neuralgia-Inducing Cavitational Osteonecrosis)The NICO lesion (Neuralgia-Inducing Cavitational Osteonecrosis, also know as Ratner's bone cavity) was first described in the dental literature in 1920 by G.V. Black. The lesion consists of ischemic osteonecrosis found in the jaws of patients with symptoms of atypical facial pain or trigeminal neuralgia. Research has shown the lesions to be difficult to diagnose. The lesion will sometimes present very subtle radiographic changes often detectable only by a technetium scan or with multiple periapical radiographs. The overlying soft tissues show no changes. Many etiologies for NICO have been suggested, but none have been substantiated through research. According to noted oral pathologist Dr. J.E. Bouquot, the typical NICO case occurs as facial pain many years after an extraction or an infection the area. Odontogenic infections and minor trauma have been suggested as initiators, and correlations to clotting or vascular abnormalities have been made based on anecdotal associations. No scientific studies have demonstrated a causative relationship between Endodontic therapy and the formation of NICO. The recommended treatment for NICO is decortication and curettage of the bony tissues. While this practice has produced relief of pain in some cases, NICO has a strong tendency to recur and to develop in other jawbone sites. Most affected sites with a postoperative NICO diagnosis have been in edentulous areas. However, some patients with long, frustrating histories of pain associated with Endodontically treated teeth have been presented the treatment option of tooth extraction followed by periapical curettage in an attempt to alleviate pain. The American Association of Endodontists cannot condone this practice when NICO is suspected. Because of the lack of clear etiological data, a NICO diagnosis should be considered only as a last resort when all possible local Odontogenic causes for facial pain have been eliminated. If a NICO lesion is suspected in relation to an Endodontically treated tooth, if possible, periradicular surgery and curettage should be attempted, not extraction. In addition, the practice of recommending the extraction of Endodontically treated teeth for the prevention of NICO, or any other disease, is unethical should be reported immediately to the appropriate state board of dentistry. The following outline provides a quick review of the steps taken in Endodontic diagnosis: I. Chief Complaint Record symptoms or problems expressed by the patient in his or her own words. II. Health History A. Medical History Take a complete medical history for each new patient. Update the medical history or each patient of record.B. Dental History Summarize present and past dental treatment. May Provide subtle clinical findings or identify source of patient's complaint Attitudes toward dental health and treatment may affect treatment planning.C. Present signs and symptoms III. Diagnostic Evaluations Subjective Examination
Tentative Diagnosis After taking histories and identifying signs and symptoms, the practitioner may reach a tentative diagnosis. The objective examination will gather the information necessary to confirm this diagnosis. Objective Examination A. Extraoral Examination Check general appearance, skin tone, and facial asymmety. Note any swelling, redness, sinus tracts, tender or enlarged lymph nodes, or tenderness or discomfort upon palpation or movement of the TMJ.B. Soft Tissue Examine the mucosa and gingiva visually and digitally for discoloration, inflammation, ulceration, swelling, and sinus tract formation.C. Dentition Examine teeth for discoloration, fracture, abrasion, erosion, caries, large restorations, discoloration or other abnormalities.D. Clinical Tests Most tests have inherent limitations. They require care on application and interpretation. The objective is to discover which tooth is different from the patient's other teeth. Always test healthy control teeth first.. .I. Periradicular Tests Percussion-a painful response is an indicator of periradicular inflammation. Palpation-same as above II. Pulp Vitality Tests These determine response to stimuli and may identify the offending tooth with an abnormal response. Always include stimuli similar to those that provoke the patient's chief complaint. 1. Cold Test Intense, prolonged pain indicates an irreversible pulpitis. Necrotic pulps do not respond. A false-negative response may occur with constricted canals. 2. Heat Test (same procedure as for cold test) Electric Pulp Testing Contrary to popular opinion and persistent notion, different response levels in electric pulp testing do not indicate different stages of pulp degeneration. Electric pulp testers do not measure the degree of health or disease of a pulp. A "yes or no" response is merely a rough indicator of the presence or absence of vital nerve tissue in the root canal system. 3. Electric Pulp Testing Before testing, clean dry, and isolate the teeth, then place a small amount of toothpaste or other conductor on the electrode. Be sure to follow your manufacturer's instructions for establishing an electrical circuit and to ensure accurate measurement with your instrument. Sensation may be described as tingling, stinging, or a feeling of heat, "fullness," or pressure.4. Cavity Test This may be helpful, especially for a tooth with a porcelain-fused-to-metal crown (PFM). Sudden, sharp sensation when the bur cuts dentin indicates that the pulp contains vital tissue.5. Peridontal Examination Periodontal probing cannot be overemphasized, since pulpal and periodontal pathosis sometimes mimic each other and must be differentiatedRadiographic Examination 1. Limitations
2. Peridacular Periradicular lesions of pulpal origin tend to have three characteristics: Loss of lamina dura apically Radiolucency remaining at the apex regardless of cone angle Radiolucency resembling a "hanging drop"A. If a radiolucency is in the periradicular region of a tooth with a vital pulp, it cannot be of pulpal origin and will be either a normal structure or another type of pathosis. B. Follow up or biopsy may be required with radiolucencies not of pulpal origin. 3. Pulpal Radiographically visible pulpal pathoses are only rarely related to irreversible pulpitis. Internal resorption or extensive diffuse calcification in the chamber may indicate long-term, low-grade irritation. "Obliteration" of canals (usually with history of trauma) does not, in itself, indicate need for treatment.Special Tests If special circumstances prevent making a definitive diagnosis, additional tests may be indicated. 1. Caries Removal In an asymptomatic vital case, caries is removed as a final test. Penetration into the pulp indicates an irreversible puplitis requiring root canal treatment.2. Selective Anesthesia Useful in painful teeth, particularly when the patient cannot isolate the offender to a specific arch.3. Transillumination For identification of vertical crown fractures, since fractured segments do not transmit the light similarly. Dark and light shadows appear at the fracture site.Analyze the Data that You Have Obtained Findings may not always be consistent, and the process of arriving at a final diagnosis depends heavily on the practitioner's critical evaluation of the findings. Formulate an Appropriate Diagnosis and Treatment Plan In addition to diagnosing pathoses and their indicated treatments, the practitioner must take into account the patient's overall needs, know the indications and contraindications for root canal therapy, and recognize those conditions that make treatment difficult.
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© 2006 Aventura Endodontic Associates, PA |
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